Scientists
at the Jawaharlal Nehru University's School of Biotechnology appointees in the
third week of January 2013 that they limit the SCO2 gene that potential
qualities of tumors. SCO2 scientists suggested that this gene may
therefore be used for the treatment of various types of cancer.
Now p53
gene was considered the proteins that block access to the tumors and prevents
thus preventing cancer. But according to the combined research carried out
by scientists at JNU School of Biotechnology and Ohio State University Medical
Center, as well as other universities, p53 gene SCO2 gene uses too much with
this tumor-suppressing quality.
In the
study of the scientific SCO2 protein injected that is encoded in SCO2-gene, in
the colon as well as the mammary tumor xenografts in mice. The result was
constant decay of tumors. When SCO2 was combined with anticancer drugs such as
tamoxifen and cisplatin, success rate hypoxic respiratory failure tumor
regression was 85 percent in four weeks.
Really,
SCO2 is responsible for enhancing the production of reactive oxygen species
(ROS), which, in turn, activates the apoptosis signal regulating kinase 1
(question 1). Apoptosis signal regulating kinase 1 (question 1), then that
happens, the growth of tumors. ROS is a kind of unstable molecule in which
oxygen, which in turn leads to easy reaction with the other molecule in the
cell. Building of ROS in the cells, can lead to damaged DNA, RNA, and proteins,
ultimately fatal. However SCO2 promote positive result of ROS cause, is
activated cell death and thus shrink the tumors.
However, scientists have also made it clear to translate these
findings to the real treatment or therapy Lane will take time. But it is still
very important step in understanding the causes of cancer agency process.
Scientists declare the importance of cancer cell metabolism and control
all relevant genes, but they need to be understood and discovered. However,
with the future of advanced therapies and treatment of cancer will not remain a
deadly disease.
